- Why I Do Take Vitamin D Supplements
- Little Known Ways Magnesium Aids Absorption of Other Nutrients
- Should You Worry if You Store Vitamin D?
- Elderly Who are in Pain Fall More
- Taking Prescription Vitamin D? You Might Want to Think Twice…
- Mothers to Be, Get Your Vitamin D in Pregnancy
- Vitamin D Rickets Is Back with a Vengeance
- Vitamin D Decreases Incidence of Blood Clots!
- Do Statins Work by Raising Vitamin D Levels?
Your Tendon, Ligament or Connective Tissue Problems Might be From Chronic Magnesium Deficiency
If you have tendon, cartilage, joint, ligament or other connective tissue problems, it’s a distinct possibility that these were caused from a chronic lack of magnesium.
I know that you probably think that you couldn’t possibly have magnesium deficiency, but if you have problems with any of these body structures it’s actually most likely that you absolutely DO have moderate and maybe even SEVERE magnesium deficiency- and that you’ve had it for many years.
The reason that you probably think that you don’t have magnesium deficiency is that you simply aren’t aware that the Signs of Magnesium Deficiency include common problems like:
- Feeling stressed
- Low back pain
And a whole host of other problems that people consider a ‘normal’ part of living in a stressful society. But these are NOT ‘normal everyday’ problems, they are distinct symptoms of magnesium deficiency that neither you nor your doctor recognizes.
Connective Tissue Synthesis
While most of the problems mentioned above can be corrected within several weeks to several months of aggressive magnesium replacement, these connective tissue problems are both less recognized as being due to magnesium deficiency as well as requiring a longer to correct, such as problems of Mitral Valve Prolapse. As a result, most people can’t imagine these problems as being caused by magnesium deficiency since the long term nature of the connective tissue degeneration makes it hard to link the two, as it might take years or even decades for this problem to manifest.
Magnesium is a critical component of collagen formation and repair, but calcium will fill up cells and ‘calcify’ cells in magnesium’s place when there is a deficiency of magnesium. This can lead to defective collagen formation over time in the presence of a chronic magnesium deficiency. One study explained it by showing that magnesium ‘hinders the mechanism by which fibroblasts (collagen creating cells) degrade defective collagen’, this is a common problem for those suffering from the Fluoroquinolone Antibiotic Toxicity, a serious and often disabling condition from antibiotics such as Levaquin and Cipro that can cause lifelong disability and pain.
In other words, when magnesium deficiency is present, the old collagen cells that need to be replaced get replaced with unhealthy calcified cells rather than flexible new healthy cells. In the case of connective tissue such as joints, ligaments and tendons, this can lead to a susceptibility to joint problems, Tendonitis, fibromyalgia (meaning literally connective tissue pain), worn cartilage, osteoarthritis and even certain heart problems that are related to connective tissue dysfunction.
Quinolone Antibiotic Toxicity
Another extremely concerning area for readers is not JUST the fact that long term magnesium deficiency can weaken connective tissue and lead to the problems above, but long-term magnesium deficiency apparently makes one more susceptible to the damaging effects of a common class of antibiotics called the Quinolone Antibiotics.
The problems that occur from these antibiotics are called Cipro Tendonitis or Quinolone Toxicity and they are of particular interest to me as I’ve done extensive research and work with people suffering from this disabling life-altering problem and have written the book The Fluoroquinolone Toxicity Solution in response to the great need for an effective solution to this problem.
One of the questions about the damage from Fluoroquinolone Antibiotic Toxicity is why certain people are more susceptible than others to the effects of these drugs. When millions take these drugs every year and only a small percentage are affected, the problem is baffling to both researchers and sufferers alike. One of the answers lies in the fact that magnesium deficiency is epidemic and long term magnesium deficiency weakens and replaces healthy connective tissue cells with calcified unhealthy cells. To show this point, a study done on rats with magnesium deficiency were given dosages of Quinolone Antibiotics and their joints were examined. (3,4)
The joints of all of the animals showed damage and swelling- regardless of whether they received the Quinolone antibiotics or not. In other words, magnesium deficiency BY ITSELF created painful and swollen joint lesions in these animals from the unhealthy calcium deposits that the magnesium deficiency allowed. The unhealthy cells created by magnesium deficiency allows small amounts of damage to the existing healthy cells become a HUGE problem of cellular death and destruction that can lead to widespread connective tissue pain and even tendon rupture with just a few doses of these toxic drugs.
While there are certainly other factors involved in being susceptible to Levaquin Tendonitis, chronic long-term magnesium deficiency is most likely one of the most predominant risk factors. Unfortunately, for various reasons, few people or their doctors are recognizing magnesium deficiency. But when you understand the powerful effects that magnesium has on the body, then you begin to see many ‘chronic health problems’ through the lens of being symptoms of the avoidable problem of magnesium deficiency which could be resolved simply by taking Magnesium Supplements. How sad that most people are not aware of this simple solution to these life-altering devastating health problems.
2) Synergistic Effect of Ofloxacin and Magnesium Deficiency on Joint Cartilage in Immature Rats
3) Integrins on joint cartilage chondrocytes and alterations by ofloxacin or magnesium deficiency in immature rats
4) Magnesium deficiency induces joint cartilage lesions in juvenile rats which are identical to quinolone-induced arthropathy